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- 2. Cellulite From a scientific point of view, the term “Cellulite” is incorrect, i.e. the suffix "ite"
- 3. Cellulite is Changes in the structural organization of the surface layers of the subcutaneous tissue, which
- 4. The following factors promote cellulite development: Lifestyle (overeating, lack of exercise, and bad habits); Circulatory disorders
- 5. Anatomy of the skin. Subcutaneous fat.
- 6. Alpha 2 receptors (lipogenesis), beta receptors (lipolysis) , Lipogenesis is accomplished by synthesis in adipocytes of
- 7. Features of fat cells In women, the number of alpha2 receptors is 6 times more in
- 8. Cellulitis Pathogenesis TSH glucagon Thyroxine ACTH vasopressin B-adrenergic receptors, G - proteins Lipogenesis (fat synthesis) Lipolysis
- 9. Pathogenesis Violation of microcirculation Accumulation of fluid in connective tissue of hypoderm Nodular sclerosis Adipocyte hypertrophy
- 10. Cellulite Classification Currently, uniform classification of cellulite does not exist. 1. According to one classification cellulite
- 11. 1. Thick cellulite - in young women with active lifestyles. Cellulite manifestations do not vary depending
- 12. Clinical Stages of Cellulite 1. Pre –cellulite stage or stage (grade) 0; 2.Primary stage or stage
- 13. Pre-cellulite Stage Dilation of small blood vessels, stagnant blood and lymph circulation Inadequate excretion, Reducing the
- 14. Primary stage Plasma and lymph accumulate in the spaces between clusters of adipocytes. Water leaves adipocytes
- 15. Micronodular Stage Fat cells are paired together in "clusters“. Vascular disorders worsen. Loose stroma turns into
- 16. Macronodular Stage Progression of circulatory disorders: venous flow is impaired, arteries are compressed hypoxia and acidosis
- 17. Methods of Cellulite Diagnostics 1. Anthropometric data. 2. Anode thermography. 3. Computed tomography and magnetic resonance
- 18. Prevention of cellulitis 1. Proper nutrition; 2. Break bad habits;. 3. Keep exercising; 4. Clothing and
- 19. Methods of Cellulite Treatment 1. Activation of beta-receptors and stimulation of lipolysis. 2. Inhibition of alpha-receptors
- 20. Drugs for Oral Use 1. Antioxidants and vitamins for cellulite treatment: E, vitamin C, A, B5,
- 21. Internal Use Preparations Bioflavonoids: 1. Means stimulating blood circulation (ginkgo biloba extract, centella asiatica, grape seed,
- 22. External Use Preparations 1. Vegetable oil for cellulite: jojoba oil, sage, evening primrose, shea butter, wheat
- 23. External Use Preparations 4. Drugs splitting fats: caffeine and other xanthine derivatives, cola extract, guarana, mate
- 24. Physical Therapeutic Methods 1. Massage; 2. Electromyostimulation; 3. Microcurrents; 4. Electrolipolysis; 5. Cavitation; 6. RF-therapy; 7.
- 30. Obesity It is a chronic recurring disease, which is accompanied by a significant increase in fat
- 35. 1. Рypodinamia. 2. Dehydration. 3. Genetic factors, in particular: Increase in activity of the enzymes of
- 36. Classification I. Primary Obesity. II. Secondary Obesity.
- 37. I. Primary Obesity. 1. Alimentary and constitutive (exogenous-constitutional); Constitutional – hereditary; 2. With eating disorders (night
- 38. II. Secondary Obesity 1. With revealed genetic defects 2. Cerebral obesity (brain tumors, skull base trauma
- 39. Stages and Types of Obesity Stages of Obesity а) progressive, б) persistent Types of Obesity 1.
- 40. Fat Can Be Distributed: 1. In the subcutaneous adipose tissue (subcutaneous fat) 2. around internal organs
- 41. Abdominal Fat
- 42. Body mass index Body mass index is the value allowing to estimate the extent of human
- 43. Four Degrees of Obesity: I degree -- body weight of the patient exceeds the normal one
- 44. Methods of General Correction Examination by an endocrinologistor gynecologist. A diet with reduced fat, carbohydrates intake.
- 45. Liposuction - operative method of vacuum liposuction after mechanical or ultrasonic disruption. In recent years, liposuction
- 46. THANKS FOR YOUR ATTENTION!
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