EKG Interpretation презентация

Содержание

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Objectives

The Basics
Interpretation
Clinical Pearls
Practice Recognition

Objectives The Basics Interpretation Clinical Pearls Practice Recognition

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The Normal Conduction System

The Normal Conduction System

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Lead Placement

aVF

Lead Placement aVF

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All Limb Leads

All Limb Leads

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Precordial Leads

Precordial Leads

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EKG Distributions

Anteroseptal: V1, V2, V3, V4
Anterior: V1–V4
Anterolateral: V4–V6, I, aVL
Lateral: I and aVL
Inferior:

II, III, and aVF
Inferolateral: II, III, aVF, and V5 and V6

EKG Distributions Anteroseptal: V1, V2, V3, V4 Anterior: V1–V4 Anterolateral: V4–V6, I, aVL

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Waveforms

Waveforms

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Interpretation

Develop a systematic approach to reading EKGs and use it every time
The

system we will practice is:
Rate
Rhythm (including intervals and blocks)
Axis
Hypertrophy
Ischemia

Interpretation Develop a systematic approach to reading EKGs and use it every time

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Rate

Rule of 300- Divide 300 by the number of boxes between each QRS

= rate

Rate Rule of 300- Divide 300 by the number of boxes between each QRS = rate

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Rate

HR of 60-100 per minute is normal
HR > 100 = tachycardia
HR < 60

= bradycardia

Rate HR of 60-100 per minute is normal HR > 100 = tachycardia HR

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Differential Diagnosis of Tachycardia

Differential Diagnosis of Tachycardia

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What is the heart rate?

(300 / 6) = 50 bpm

www.uptodate.com

What is the heart rate? (300 / 6) = 50 bpm www.uptodate.com

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Rhythm

Sinus
Originating from SA node
P wave before every QRS
P wave in same direction

as QRS

Rhythm Sinus Originating from SA node P wave before every QRS P wave

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What is this rhythm?

Normal sinus rhythm

What is this rhythm? Normal sinus rhythm

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Normal Intervals

PR
0.20 sec (less than one large box)
QRS
0.08 – 0.10 sec (1-2 small

boxes)
QT
450 ms in men, 460 ms in women
Based on sex / heart rate
Half the R-R interval with normal HR


Normal Intervals PR 0.20 sec (less than one large box) QRS 0.08 –

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Prolonged QT

Normal
Men 450ms
Women 460ms
Corrected QT (QTc)
QTm/√(R-R)
Causes
Drugs (Na channel blockers)
Hypocalcemia, hypomagnesemia, hypokalemia
Hypothermia
AMI
Congenital
Increased

ICP

Prolonged QT Normal Men 450ms Women 460ms Corrected QT (QTc) QTm/√(R-R) Causes Drugs

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Blocks

AV blocks
First degree block
PR interval fixed and > 0.2 sec
Second degree

block, Mobitz type 1
PR gradually lengthened, then drop QRS
Second degree block, Mobitz type 2
PR fixed, but drop QRS randomly
Type 3 block
PR and QRS dissociated

Blocks AV blocks First degree block PR interval fixed and > 0.2 sec

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What is this rhythm?

First degree AV block PR is fixed and longer

than 0.2 sec

What is this rhythm? First degree AV block PR is fixed and longer than 0.2 sec

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What is this rhythm?

Type 1 second degree block (Wenckebach)

What is this rhythm? Type 1 second degree block (Wenckebach)

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What is this rhythm?

Type 2 second degree AV block Dropped QRS

What is this rhythm? Type 2 second degree AV block Dropped QRS

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What is this rhythm?

3rd degree heart block (complete)

What is this rhythm? 3rd degree heart block (complete)

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The QRS Axis

Represents the overall direction of the heart’s activity
Axis of

–30 to +90 degrees is normal

The QRS Axis Represents the overall direction of the heart’s activity Axis of

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The Quadrant Approach

QRS up in I and up in aVF = Normal

The Quadrant Approach QRS up in I and up in aVF = Normal

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What is the axis?

Normal- QRS up in I and aVF

What is the axis? Normal- QRS up in I and aVF

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Hypertrophy

Add the larger S wave of V1 or V2 in mm, to the

larger R wave of V5 or V6.
Sum is > 35mm = LVH

Hypertrophy Add the larger S wave of V1 or V2 in mm, to

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Ischemia

Usually indicated by ST changes
Elevation = Acute infarction
Depression = Ischemia
Can manifest as T

wave changes
Remote ischemia shown by q waves

Ischemia Usually indicated by ST changes Elevation = Acute infarction Depression = Ischemia

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What is the diagnosis?

Acute inferior MI with ST elevation in leads II,

III, aVF

What is the diagnosis? Acute inferior MI with ST elevation in leads II, III, aVF

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What do you see in this EKG?

ST depression II, III, aVF, V3-V6 =

ischemia

What do you see in this EKG? ST depression II, III, aVF, V3-V6 = ischemia

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Let’s Practice

The sample EKGs were obtained from the following text:

Let’s Practice The sample EKGs were obtained from the following text:

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Normal Sinus Rhythm

Mattu, 2003

Normal Sinus Rhythm Mattu, 2003

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First Degree Heart Block

PR interval >200ms

First Degree Heart Block PR interval >200ms

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Accelerated Idioventricular

Ventricular escape rhythm, 40-110 bpm
Seen in AMI, a marker of reperfusion

Accelerated Idioventricular Ventricular escape rhythm, 40-110 bpm Seen in AMI, a marker of reperfusion

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Junctional Rhythm

Rate 40-60, no p waves, narrow complex QRS

Junctional Rhythm Rate 40-60, no p waves, narrow complex QRS

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Hyperkalemia

Tall, narrow and symmetric T waves

Hyperkalemia Tall, narrow and symmetric T waves

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Wellen’s Sign

ST elevation and biphasic T wave in V2 and V3
Sign of large

proximal LAD lesion

Wellen’s Sign ST elevation and biphasic T wave in V2 and V3 Sign

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Brugada Syndrome

RBBB or incomplete RBBB in V1-V3 with convex ST elevation

Brugada Syndrome RBBB or incomplete RBBB in V1-V3 with convex ST elevation

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Brugada Syndrome

Autosomal dominant genetic mutation of sodium channels
Causes syncope, v-fib, self terminating VT,

and sudden cardiac death
Can be intermittent on EKG
Most common in middle-aged males
Can be induced in EP lab
Need ICD

Brugada Syndrome Autosomal dominant genetic mutation of sodium channels Causes syncope, v-fib, self

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Premature Atrial Contractions

Trigeminy pattern

Premature Atrial Contractions Trigeminy pattern

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Atrial Flutter with Variable Block

Sawtooth waves
Typically at HR of 150

Atrial Flutter with Variable Block Sawtooth waves Typically at HR of 150

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Torsades de Pointes

Notice twisting pattern
Treatment: Magnesium 2 grams IV

Torsades de Pointes Notice twisting pattern Treatment: Magnesium 2 grams IV

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Digitalis


Dubin, 4th ed. 1989

Digitalis Dubin, 4th ed. 1989

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Lateral MI

Reciprocal changes

Lateral MI Reciprocal changes

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Inferolateral MI

ST elevation II, III, aVF
ST depression in aVL, V1-V3 are reciprocal changes


Inferolateral MI ST elevation II, III, aVF ST depression in aVL, V1-V3 are reciprocal changes

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Anterolateral / Inferior Ischemia

LVH, AV junctional rhythm, bradycardia

Anterolateral / Inferior Ischemia LVH, AV junctional rhythm, bradycardia

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Left Bundle Branch Block

Monophasic R wave in I and V6, QRS > 0.12

sec
Loss of R wave in precordial leads
QRS T wave discordance I, V1, V6
Consider cardiac ischemia if a new finding

Left Bundle Branch Block Monophasic R wave in I and V6, QRS >

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Right Bundle Branch Block

V1: RSR prime pattern with inverted T wave
V6: Wide deep

slurred S wave

Right Bundle Branch Block V1: RSR prime pattern with inverted T wave V6:

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First Degree Heart Block, Mobitz Type I (Wenckebach)

PR progressively lengthens until QRS drops

First Degree Heart Block, Mobitz Type I (Wenckebach) PR progressively lengthens until QRS drops

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Supraventricular Tachycardia

Narrow complex, regular; retrograde P waves, rate <220

Retrograde P waves

Supraventricular Tachycardia Narrow complex, regular; retrograde P waves, rate Retrograde P waves

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Right Ventricular Myocardial Infarction

Found in 1/3 of patients with inferior MI
Increased morbidity and

mortality
ST elevation in V4-V6 of Right-sided EKG

Right Ventricular Myocardial Infarction Found in 1/3 of patients with inferior MI Increased

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Ventricular Tachycardia

Ventricular Tachycardia

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Prolonged QT

QT > 450 ms
Inferior and anterolateral ischemia

Prolonged QT QT > 450 ms Inferior and anterolateral ischemia

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Second Degree Heart Block, Mobitz Type II

PR interval fixed, QRS dropped intermittently

Second Degree Heart Block, Mobitz Type II PR interval fixed, QRS dropped intermittently

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Acute Pulmonary Embolism

SIQIIITIII in 10-15%
T-wave inversions, especially occurring in
inferior

and anteroseptal simultaneously
RAD

Acute Pulmonary Embolism SIQIIITIII in 10-15% T-wave inversions, especially occurring in inferior and anteroseptal simultaneously RAD

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Wolff-Parkinson-White Syndrome

Short PR interval <0.12 sec
Prolonged QRS >0.10 sec
Delta wave
Can simulate ventricular hypertrophy,

BBB and previous MI

Wolff-Parkinson-White Syndrome Short PR interval Prolonged QRS >0.10 sec Delta wave Can simulate

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Hypokalemia

U waves
Can also see PVCs, ST depression, small T waves

Hypokalemia U waves Can also see PVCs, ST depression, small T waves

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